On the contrary, it stops the cravings. And you're full for longer as it slows down the passage of food in your system.

Of course, it is up to the user to 1) exercise and 2) eat healthy.

Depends for whom, some here seem to continue having cravings, see other comments.

if you have paid attention to any article in the last year, one of the primary functions of these drugs is reducing the cravings.

To all food or just bad food? And how does it do that?

I saw in this thread that it reduces cravings to fatty food which is bad news.

There's a lot of information you can find if you spend some time to do the research before making declarations on the topic, but studies have shown significant reductions in alcohol consumption, for one thing. Anecdotally, multiple people I know who are on GLP-1 agonists have said that they feel much less of a desire to drink and have much less when they do.

I believe the anecdotes I've seen were about people on this drug finding that they thought about formerly favorite unhealthy foods and viscerally recoiled at the idea of eating them.

My speculation is that it's a gut biota change driving the change in cravings, but I'm not sure there's much research on the mechanisms involved yet, since these drugs are very new, comparatively. A very small study apparently suggested it ameliorated some of the numbness to intense flavors that the obese have been shown to have in previous studies, so maybe I'm completely off base.